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Etifoxine: Difference between revisions

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[http://www.ncbi.nlm.nih.gov/pmc/articles/PMC4364751/pdf/pone.0120473.pdf do Rego JL, Vaudry D, Vaudry H (2015) The Non-Benzodiazepine Anxiolytic Drug Etifoxine Causes a Rapid, Receptor-Independent Stimulation of Neurosteroid Biosynthesis. PLoS ONE 10(3): e0120473. doi:10.1371/journal.pone.0120473]
[http://www.ncbi.nlm.nih.gov/pmc/articles/PMC4364751/pdf/pone.0120473.pdf do Rego JL, Vaudry D, Vaudry H (2015) The Non-Benzodiazepine Anxiolytic Drug Etifoxine Causes a Rapid, Receptor-Independent Stimulation of Neurosteroid Biosynthesis. PLoS ONE 10(3): e0120473. doi:10.1371/journal.pone.0120473]
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[[Category:GABAA receptor positive allosteric modulators]]

Revision as of 19:09, 5 August 2016

Choi, YM, and Kim, KH (2015). Etifoxine for Pain Patients with Anxiety

etifoxine is an anticonvulsant and ansiolytic agent. It is not recommended under the age of 18 years.[1]

The known mechanism oof action is potentiation of GABAA receptor, via stimulation of neurosteroid production. The etifoxine therapy promoted the regeneration of axons [2] [3]

Links

Etifoxine promotes glial‑derived neurotrophic factor‑induced neurite outgrowth in PC12 cells

do Rego JL, Vaudry D, Vaudry H (2015) The Non-Benzodiazepine Anxiolytic Drug Etifoxine Causes a Rapid, Receptor-Independent Stimulation of Neurosteroid Biosynthesis. PLoS ONE 10(3): e0120473. doi:10.1371/journal.pone.0120473

  1. ↑ https://www.awakebrain.com/stresam-etifoxine
  2. ↑ Etifoxine improves peripheral nerve regeneration and functional recovery
  3. ↑ Etifoxine for Pain Patients with Anxiety
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